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    GENE:

    ATP5F1E (ATP Synthase F1 Subunit Epsilon)

    i
    Other names: ATP Synthase F1 Subunit Epsilon, ATP Synthase, H+ Transporting, Mitochondrial F1 Complex, Epsilon Subunit, ATP Synthase Subunit Epsilon, Mitochondrial, ATP5E, Mitochondrial ATP Synthase Epsilon Chain, H(+)-Transporting Two-Sector ATPase, ATPase Subunit Epsilon, Mitochondrial ATPase, F(0)F(1)-ATPase, ATP5F1E, MC5DN3, ATPE
    Associations

    News

    Trials
    7ms
    Distinctive chromosomal, mutational and transcriptional profiling in colon versus rectal cancers. (PubMed, J Transl Med)
    Collectively, our findings provide robust molecular evidence that CC and RC follow divergent oncogenic pathways, emphasizing the need for site-specific biomarker development and therapeutic targeting in colorectal cancer.
    Clinical • Journal • Tumor mutational burden
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    BRAF (B-raf proto-oncogene) • TP53 (Tumor protein P53) • PIK3CA (Phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha) • TMB (Tumor Mutational Burden) • NRAS (Neuroblastoma RAS viral oncogene homolog) • GATA2 (GATA Binding Protein 2) • EHD1 (EH Domain Containing 1) • ATP5F1E (ATP Synthase F1 Subunit Epsilon) • HSPD1 (Heat Shock Protein Family D (Hsp60) Member 1) • SETD1A (SET Domain Containing 1A) • ACVR1B (Activin A Receptor Type 1B)
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    TP53 mutation • BRAF mutation • NRAS mutation • PIK3CA mutation
    almost3years
    Hsa_circ_0079480 enhances cell proliferation, migration, and invasion in colorectal cancer through miR-498/ATP5E axis. (PubMed, Kaohsiung J Med Sci)
    ATP5E overexpression mitigated the inhibitory effect of hsa_circ_0079480 on CRC cell malignant behaviors. Since hsa_circ_0079480 knockdown inhibited CRC cells malignant behaviors through regulation of the miR-498/ATP5E axis, it can be concluded that hsa_circ_0079480 might have great potential as therapeutic target for CRC.
    Journal
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    ATP5F1E (ATP Synthase F1 Subunit Epsilon)